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A Unique Type of
Albert Enayati, B.S., Ch.E., M.S.M.E.
Lyn Redwood, R.N., M.S.N., C.R.N.P.
Woody McGinnis, M.D.
2000 by ARC Research
14 Commerce Drive
Cranford, NJ 07016
April 3, 2000
Autism is a neurodevelopmental syndrome characterized by impairments in social relatedness, language, and communication, a need for routine and sameness, abnormal movements, and sensory dysfunction. Mercury is a toxic metal that can exist as a pure element or in a variety of inorganic and organic forms and can cause immune, sensory, neurological, motor, and other behavioral dysfunctions.
The characteristics of autism and mercury poisoning, derived from a review of medical literature, have been found, upon comparison, to be strikingly similar. The characteristics of both disorders are summarized in the following table and fully elucidated in the body of this document. The parallels between the two diseases are so close that it would be unreasonable to assume that the similarities occur by chance.
We claim that autism is a form of mercury poisoning, based on similarities of characteristics and on the known exposure to mercury of the majority of US children. The exposure route is childhood vaccines, most of which contain thimerosal, a preservative comprised of 50% ethylmercury by weight. The amount of mercury a typical child under two years receives from vaccinations equates to 237.5 micrograms, or 3.53 x 1017 molecules (353,000,000,000,000,000 molecules), most of which is not excreted and goes directly to the brain. The amount is known to exceed Federal safety standards, but is still considered a “low” level, such that only a small percentage of exposed individuals will exhibit signs of toxicity. Affected individuals are those genetically prone to mercury sensitivity, which is consistent with the observed high heritability rate of autism. Furthermore, the timing of mercury exposure via vaccines coincides with the emergence of autistic symptoms. Moreover, mercury has been detected in urine, hair, and blood samples from autistic children, and parental reports, though limited at this date, indicate significant improvement in symptoms with administration of standard heavy metal chelators. Thus, the four agreed-upon criteria used by clinicians to diagnose mercury poisoning – i.e., observable symptoms, known exposure at the time of symptom onset, detectable levels in biologic samples, and improvement with chelation - have been met for autism.
The phenotypic expression of mercury poisoning varies by a host of factors – including type of mercury given, method of administration, rate and level of dose, individual genotype, and age of patient – so that each variation in factors has created in the past a slightly different manifestation of the disease – Mad Hatter’s disease, Minamata disease, and acrodynia, for example. The pathology arising from the set of mercury-related variables involved in autism – intermittent bolus doses of ethylmercury injected into genetically susceptible infants and toddlers – has never been reported before in medical literature. Thus we argue that autism represents a unique form of mercury poisoning not heretofore described. Our findings have widespread implications for the affected population of autistic individuals, for other unexplained disorders with symptoms similar to heavy metal intoxication, and for childhood vaccination programs.
Summary Comparison of Characteristics
of Autism & Mercury Poisoning
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