George R. Jackson, M. D., Ph. D




НазваниеGeorge R. Jackson, M. D., Ph. D
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Дата27.09.2012
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ТипДокументы
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Inactivation of Drosophila Apaf-1 blocks polyglutamine-induced neurodegeneration. Proc. Soc. Neurosci. 29: 130.4




  1. Jackson, G.R., and Sang, T.-K. (2004): Dopaminergic neurodegeneration and parkinsonism induced by mutant human parkin in Drosophila. 56th annual meeting, American Academy of Neurology, San Francisco.




  1. Sang, T.-K., Li, C.J., Abrams, J.M., Zipursky, S.L., and Jackson, G.R. (2004): Inactivation of Drosophila Apaf -1 blocks polyglutamine pathogenesis: Implications for Huntington’s disease. Movement Disorders (2004) 19: supplement 9: S31. The Movement Disorder Society's 8th International Congress on Parkinson's Disease and Movement Disorders, Rome.




  1. Jackson, G.R., and Sang, T.-K. (2004): Puromycin-sensitive aminopeptidase regulates tauopathy in vivo. Neurobiol Aging 25: S43. The 9th International Conference on Alzheimer’s Disease and Related Disorders, Philadelphia.




  1. Sang, T.-K., Chang, H.-Y. Krantz, D.E., and Jackson, G.R. (2004): Role of dopamine in mutant human parkin-induced neurodegeneration and motor deficits in Drosophila. Proc. Soc. Neurosci. 30: 140.3.




  1. Jackson, G.R., Sang, T.-K., Ko, J., Khoshnan, A., and Patterson, P.H. (2004): Inhibition of mutant huntingtin-induced neurodegeneration in vivo by expression of a polyproline-binding single chain antibody. Proc. Soc. Neurosci. 30: 938.5.




  1. Chang, H.-Y., Sang, T.-K., Jackson, G.R., and Krantz, D.E. (2004): A screen for mutants enhancing dopamine and serotonin transmission. Proc. Soc. Neurosci. 30: 1024.17




  1. Mee, L., Honkala, H., Kestila, M., Finnila, S., Visapaa, I., Hovanes, K., Lee, J., Salonen, R., Jackson, G., and Peltonen, L. (2004): Identification and characterization of the hydrolethalus syndrome gene. Am. Soc. Hum. Genet. 1977:F3.




  1. Karsten, S.L., Sang, T.-K., Liu, J.K., Chatterjee, S., Gehman, L.T., Pomakian, J., Oh, H.Y., Vinters, H.V., Geschwind, D.H., and Jackson, G.R. (2005): Microarray and functional analysis identifies puromycin-sensitive aminopeptidase (PSA) as a novel neuroprotective agent in tau-induced neurodegeneration. Proc. Soc. Neurosci. 31: 473.12.




  1. LaVoie, M.J., Sang, T.-K., Cortese, G., Chang, H-Y., Krantz, D.E., and Jackson, G.R., (2006): Dopamine and parkin ionteractions in vitro and in vivo: A novel model of selective dopaminergic regeneration in Drosophila. First World Parkinson Congress, Washington, D.C. (selected for poster tour).




  1. Chatterjee, S., and Jackson, G.R. (2007): Regulation of tau phosphorylation and toxicity: Insights from a Drosophila model. Eighth International Congress on Alzheimer’s and Parkinson’s Diseases, Salzburg, Austria. Neurobiol Disease 4 (s1): 156.




  1. Sang, T.-K., Chang, H.Y., Krantz, D.E., and Jackson, G.R. (2007): The human parkin mutation T240R causes selective loss of dopaminergic neurons in a Drosophila model. Eighth International Congress on Alzheimer’s and Parkinson’s Diseases, Salzburg, Austria. Neurobiol Disease 4 (suppl 1): 246.




  1. Ratnaparkhi, A., Lawless, G.M., and Jackson, G.R. (2007): A Drosophila model for motor neuron disease caused by a mutation in VAPB/DVAP33A: Evidence for a dominant negative mechanism. 12th biennial Neurobiol. Drosophila Meeting, Cold Spring Harbor, N.Y.




  1. Ratnaparkhi, A., Lawless, G.M., Schweizer, F.E., Golshani, P., and Jackson, G.R. (2008): A Drosophila model of ALS: Human ALS-associated mutation in VAP33A suggests a dominant negative mechanism. Proc. Soc. Neurosci. 32: 212.11.



INVITED LECTURES


  1. University of Chicago, Department of Neurology; Dec. 7, 2000; Molecular Genetics of Neurodegeneration”




  1. Parkinson’s Institute, Sunnyvale, CA; July 5, 2001; Molecular Genetics of Neurodegeneration”




  1. Oregon Health and Sciences University, Department of Neurology; July 26, 2001; “Molecular Genetics of Neurodegeneration”




  1. University of Toronto, Centre for Research in Neurodegenerative Diseases; Sept. 5, 2001; “Molecular Genetics of Neurodegeneration”




  1. Toronto Western Hospital, Sept. 6, 2001; “Update on the Pathogenesis of Glutamine Repeat Disorders”




  1. Palo Alto VA Hospital, Geriatric Research, Education, and Clinical Component; October 26, 2001; “Molecular Genetics of Neurodegeneration”




  1. Vanderbilt University, Department of Neurology; Feb. 8, 2002; “Molecular Genetics of Neurodegeneration”




  1. Genome Institute of Singapore; May 15, 2002; “Molecular Genetics of Neurodegeneration”




  1. University of Toronto, Centre for Research in Neurodegenerative Diseases; June 10,

2002; “Update on the Pathogenesis of Polyglutaminopathies and Tauopathies”


  1. University of Texas Medical Branch at Galveston, Department of Neurology; Sept. 4,

2002; “Molecular Genetics of Neurodegeneration”


  1. University of Toronto, Centre for Research in Neurodegenerative Diseases; Sept. 5, 2001; “Molecular Genetics of Neurodegeneration”




  1. VA/UCLA Research Conference on Parkinson’s and Movement Disorders, Los Angeles, CA; Sept. 10, 2002; “Fruit Fly Models of Neurodegeneration”




  1. Pfizer Global Research and Development, Groton, CT; Oct. 15, 2002; “Drosophila Models of Tauopathies”




  1. Elan Pharmaceuticals, South San Francisco, CA; Dec. 11, 2002; “Modeling Neurofibrillary Pathology using Human Tau in Drosophila”




  1. University of California, San Francisco, Department of Neurology; Jan. 15, 2003;“The Eye of the Fly: Visualizing Genetic Modifiers for Neurodegenerative Diseases”




  1. 8TH Annual UCLA Research Conference on Aging; June 25, 2003; “Lessons About the Role of Tau In Alzheimer’s Disease from the Fruit Fly”




  1. EnVivo Pharmaceuticals, Cambridge, MA; Sept. 23, 2003; “Role of Apaf-1 in Polyglutamine Pathogenesis”




  1. American Association of Geriatric Psychiatry, Symposium: Agitation: From Neurobiology to Treatment, Baltimore, MD; Feb. 22, 2004; “Examining Neuroprotective Strategies Using A Fly Transgenic Bioassay”




  1. University of Texas Southwestern Medical School at Dallas, Dallas, TX, Center for Basic Neuroscience; June 21, 2004;“Identifying Potential Therapeutic Targets for Human Neurodegenerative Diseases Using Drosophila Genetics”




  1. Cedars-Sinai Medical Center, Neurology Grand Rounds, Los Angeles, CA; June 23, 2004; “Role of Apaf-1 in Polyglutamine Pathogenesis: Implications for Huntington’s Disease”




  1. Johns Hopkins University School of Medicine, Department of Neuroscience, November 10, 2005;“Identification of a Tau Aminopeptidase that Inhibits Neurodegeneration”




  1. Emory University Center for Neurodegenerative Disease, December 9, 2005; “Dominant Phenotypes of Human Parkin Mutations: A New Model for Parkinson’s Disease in Drosophila




  1. University of Missouri, Kansas City, School of Biological Sciences, September 7, 2006; “Tangling with Tau using Drosophila”




  1. University of Washington, Center for Neurogenetics and Neurotherapeutics, December 14, 2006; “Untangling Tauopathy: Lessons from a Drosophila Model”




  1. Vanderbilt University Department of Biology, March 19, 2009, "Molecular Genetics of Neurodegeneration from the Fly’s Viewpoint




  1. Mitchell Center, March 30, 2009, “Molecular Genetics of ALS8 (Lou Gehrig’s Disease):




  1. Neuroscience Graduate Program, May 20, 2009: “Neurodegenerative Tauopathies”




  1. Center for Research on Occupational and Environmental Toxicology, Oregon Health and Sciences University, June 1, 2009, “Neurodegenerative Tauopathies.

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